Methamphetamine exposure induces neuropathic protein β-Amyloid expression-Reference-Cited by-同舟云学术

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Methamphetamine exposure induces neuropathic protein β-Amyloid expression

Published:2019-02 Issue: Volume:54 Page:304-309
ISSN:0887-2333
Container-title:Toxicology in Vitro
language:en
Short-container-title:Toxicology in Vitro

Author:

Chen Lingling,Yu Pengfei,Zhang Li,Zou Yuxia,Zhang Yujuan,Jiang Lei,Gao Rong,Xiao Hang,Qian Yanning,Wang Jun

Funder

National Natural Science Foundation of China

Natural Science Foundation of Jiangsu Province

Key disease of Jiangsu Province Science and Technology Department

Nanjing Medical University

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference38 articles.

1. Methamphetamine inhibits the glucose uptake by human neurons and astrocytes: stabilization by acetyl-L-carnitine;Abdul;PLoS One,2011

2. The role of dopamine receptors in the neurotoxicity of methamphetamine;Ares-Santos;J. Intern. Med.,2013

3. Etiology and pathogenesis of late-onset Alzheimer's disease;Balin;Curr Allergy Asthma Rep,2014

4. Chronic methamphetamine self-administration disrupts cortical control of cognition;Bernheim;Neurosci. Biobehav. Rev.,2016

5. BACE1 is the major beta-secretase for generation of Abeta peptides by neurons;Cai;Nat. Neurosci.,2001

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1. DReAmocracy: A Method to Capitalise on Prior Drug Discovery Efforts to Highlight Candidate Drugs for Repurposing;International Journal of Molecular Sciences;2024-05-13

2. Role of the Peli1–RIPK1 Signaling Axis in Methamphetamine-Induced Neuroinflammation;ACS Chemical Neuroscience;2023-02-10

3. Melatonin Attenuates Methamphetamine-Induced Alteration of Amyloid β Precursor Protein Cleaving Enzyme Expressions via Melatonin Receptor in Human Neuroblastoma Cells;Neurotoxicity Research;2022-06-01

4. Targeting Aβ and p-Tau Clearance in Methamphetamine-Induced Alzheimer’s Disease-Like Pathology: Roles of Syntaxin 17 in Autophagic Degradation in Primary Hippocampal Neurons;Oxidative Medicine and Cellular Longevity;2022-05-18

5. Ketamine promotes the amyloidogenic pathway by regulating endosomal pH;Toxicology;2022-04

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