Herpes Virus Entry Mediator (HVEM): A Novel Potential Mediator of Trauma-Induced Immunosuppression
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Surgery
Reference49 articles.
1. The systemic immune response to trauma: an overview of pathophysiology and treatment;Lord;Lancet,2014
2. Failure to normalize lymphopenia following trauma is associated with increased mortality, independent of the leukocytosis pattern;Heffernan;Crit Care,2012
3. Relationship between T lymphocyte apoptosis and anergy following trauma;Pellegrini;J Surg Res,2000
4. Innate immunity in the persistent inflammation, immunosuppression and catabolism syndrome and its implications for therapy;Horiguchi;Front Immunol,2018
5. Lymphtoxin beta receptor-Ig protectes from T-cell-mediated liver injury in mice through blocking LIGHT/HVEM signaling;An;Biol Pharm Bull,2006
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1. Etiological characteristics of wound infection in severe trauma patients and logistic regression analysis of influencing factors of infection;International Wound Journal;2024-01-31
2. Pneumonia in the first week after polytrauma is associated with reduced blood levels of soluble herpes virus entry mediator;Frontiers in Immunology;2023-12-22
3. VISTA nonredundantly regulates proliferation and CD69low γδ T cell accumulation in the intestine in murine sepsis;Journal of Leukocyte Biology;2023-11-30
4. Lymphocyte HVEM/BTLA co-expression after critical illness demonstrates severity indiscriminate upregulation, impacting critical illness-induced immunosuppression;Frontiers in Medicine;2023-05-25
5. The role of the BTLA-HVEM complex in the pathogenesis of autoimmune diseases;Cellular Immunology;2022-06
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