Oral administration of NO synthase inhibitor failed to promote arteriosclerotic lesions in the aorta and the coronary arteries of rabbits fed cholesterol
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference15 articles.
1. Is NO an endogenous antiatherogenic molecule? (Editorial);Cooke;Arterioscler. Thromb.,1994
2. Long-term inhibition of NO synthesis promotes atherosclerosis in the hypercholesterolemic rabbit thoracic aorta. PGH2 does not contribute to impaired endothelium-dependent relaxation;Naruse;Arterioscler. Thromb.,1994
3. Chronic inhibition of nitric oxide production accelerates neointima formation and impairs endothelial function in hypercholesterolemic rabbits;Cayatte;Arterioscler. Thromb.,1994
4. Chronic inhibition of nitric oxide synthesis causes coronary microvascular remodeling in rats;Numaguchi;Hypertension,1995
5. Causal relationship between occlusive lesions of the coronary artery and myocardial fibrosis in arteriosclerotic rabbits—differences between cholesterol-fed and heritable hyperlipidemic rabbits;Nakamura;Atherosclerosis,1996
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