Mycoplasma arthritidis superantigen (MAM)-induced macrophage nitric oxide release is MHC class ii restricted, interferonγ dependent, and toll-like receptor 4 independent

Author:

Ribeiro-Dias Fátima,Shio Marina Tiemi,Timenetsky Jorge,Oliane Ana Paula Camilo,Metran Camila Cardoso,Pessoa Fábio Borges,Jancar Sonia

Publisher

Elsevier BV

Subject

Cell Biology

Reference55 articles.

1. Mycoplasma-induced arthritis in animals;Cole;Rev. Rheum.,1999

2. Anti-MAM antibodies in rheumatic disease;Sawitzke;J. Rheumatol.,2000

3. Stimulation of mouse lymphocytes by a mitogen derived from Mycoplasma arthritidis (MAM). VIII. Selective activation of T cells expressing distinct Vβ T cell receptors from various strains of mice by the “superantigen” MAM;Cole;J. Immunol.,1990

4. Interaction of Mycoplasma arthritidis superantigen with human T cells;Posnett;Clin. Infect. Dis.,1993

5. Limited T-cell receptor β-chain heterogeneity among interleukin 2 receptor-positive synovial T cells suggests a role for superantigen in rheumatoid arthritis;Howell;Proc. Natl. Acad. Sci. USA,1991

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2. Coexpression of TLR2 or TLR4 with HLA-DR Potentiates the Superantigenic Activities ofMycoplasma arthritidis–Derived Mitogen;The Journal of Immunology;2014-02-03

3. Staphylococcal and Streptococcal Superantigens;Biodefense Research Methodology and Animal Models, Second Edition;2012-02-07

4. Mycoplasma;Topley & Wilson's Microbiology and Microbial Infections;2010-03-15

5. Crucial cytokine interactions in nitric oxide production induced by Mycoplasma arthritidis superantigen;Microbes and Infection;2008-11

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