Inactivation of the p53 gene by either mutation or HPV infection is extremely frequent in human oral squamous cell carcinoma cell lines-Reference-Cited by-同舟云学术

Inactivation of the p53 gene by either mutation or HPV infection is extremely frequent in human oral squamous cell carcinoma cell lines

Published:1994-01 Issue:5 Volume:30 Page:338-345
ISSN:0964-1955
Container-title:European Journal of Cancer Part B: Oral Oncology
language:en
Short-container-title:European Journal of Cancer Part B: Oral Oncology

Author:

Min Byung-Moo,Baek Jeong-Hwa,Shin Ki-Hyuk,Gujuluva Chandrasekhar N.,Cherrick Henry M.,Park No-Hee

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference47 articles.

1. Wild-type p53 can inhibit oncogene-mediated focus formation;Eliyahu,1989

2. Growth suppression induced by wild-type p53 protein is accompanied by selective down-regulation of proliferating-cell nuclear antigen expression;Mercer,1991

3. High incidence of lung, bone, and lymphoid tumors in transgenic mice overexpressing mutant alleles of the p53 oncogene;Lavigueur;Mol Cell Biol,1989

4. Frequent mutation of p53 gene in human esophageal cancer;Hollstein,1990

5. p53 Mutations in human cancers;Hollstein;Science,1991

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