Sympathetic activity induced by naloxone-precipitated morphine withdrawal is blocked in genetically engineered mice lacking functional CRF1 receptor-Reference-Cited by-同舟云学术

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Sympathetic activity induced by naloxone-precipitated morphine withdrawal is blocked in genetically engineered mice lacking functional CRF1 receptor

Published:2015-02 Issue:1 Volume:283 Page:42-49
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

García-Carmona Juan-Antonio,Martínez-Laorden Elena,Milanés María-Victoria,Laorden María-Luisa

Funder

Ministerio de Ciencia e Innovación

Red de trastornos adictivos

Instituto de Salud Carlos III

Fundación Séneca

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference63 articles.

1. Ventrolateral medullary control of cardiovascular activity during muscle contraction;Ally;Neurosci. Biobehav. Rev.,1998

2. The PKs PKA and ERK 1/2 are involved in phosphorylation of TH at Serine 40 and 31 during morphine withdrawal in rat hearts;Almela;Br. J. Pharmacol.,2008

3. Central stresscopin modulates cardiovascular function through the adrenal medulla in conscious rats;Chu;Regul. Pept.,2004

4. Gender-linked differences in the expression of physical dependence in the rat;Cicero;Pharmacol. Biochem. Behav.,2002

5. Spontaneous withdrawal in opiate-dependent Fischer 344, Lewis and Sprague–Dawley rats;Cobuzzi;Pharmacol. Biochem. Behav.,2011

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4. Methamphetamine withdrawal induces activation of CRF neurons in the brain stress system in parallel with an increased activity of cardiac sympathetic pathways;Naunyn-Schmiedeberg's Archives of Pharmacology;2018-01-30

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