ATPR-induced differentiation and G0/G1 phase arrest in acute promyelocytic leukemia by repressing EBP50/NCF1 complex to promote the production of ROS-Reference-Cited by-同舟云学术

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ATPR-induced differentiation and G0/G1 phase arrest in acute promyelocytic leukemia by repressing EBP50/NCF1 complex to promote the production of ROS

Published:2019-09 Issue: Volume:379 Page:114638
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Feng Yubin,Niu Ruowen,Cheng Xin,Wang Ke,Du Yan,Peng Xiaoqing,Chen Feihu

Funder

National Major Scientific and Technological Special Project

National Science Foundation of China

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference41 articles.

1. Binding of EBP50 to Nox organizing subunit p47phox is pivotal to cellular reactive species generation and altered vascular phenotype;Al Ghouleh;Proc. Natl. Acad. Sci. U. S. A.,2016

2. The ROS/JNK/ATF2 pathway mediates selenite-induced leukemia NB4 cell cycle arrest and apoptosis in vitro and in vivo;An;Cell Death Dis.,2013

3. ARHGAP12 functions as a developmental brake on excitatory synapse function;Ba;Cell Rep.,2016

4. The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology;Bedard;Physiol. Rev.,2007

5. NHERF1/EBP50 in breast cancer: clinical perspectives;Bellizzi;Breast care (Basel),2010

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