Polydatin inhibits proliferation and promotes apoptosis of doxorubicin-resistant osteosarcoma through LncRNA TUG1 mediated suppression of Akt signaling-Reference-Cited by-同舟云学术

Polydatin inhibits proliferation and promotes apoptosis of doxorubicin-resistant osteosarcoma through LncRNA TUG1 mediated suppression of Akt signaling

Published:2019-05 Issue: Volume:371 Page:55-62
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Hu Tongzhou,Fei Zhenghua^ORCID,Su Huafang,Xie Raoying,Chen Liting

Funder

Major Disease Control Science and Technology Action Plan - Trauma Repair Special

Ningbo Natural Science Foundation

Xiangshan Science and Technology Project

Natural Science Foundation of China

Natural Science Foundation of Zhejiang

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference46 articles.

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2. An integrated genome-wide CRISPRa approach to functionalize lncRNAs in drug resistance;Bester;Cell,2018

3. Mitochondria-targeted doxorubicin: a new therapeutic strategy against doxorubicin-resistant osteosarcoma;Buondonno;Mol. Cancer Ther.,2016

4. Polydatin-induced cell apoptosis and cell cycle arrest are potentiated by Janus kinase 2 inhibition in leukemia cells;Cao;Mol. Med. Rep.,2016

5. Polydatin down-regulates the phosphorylation level of Creb and induces apoptosis in human breast cancer cell;Chen;PLoS One,2017

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