Exposure to bisphenol A in young adult mice does not alter ovulation but does alter the fertilization ability of oocytes
Author:
Funder
Consejo Nacional de Ciencia y Tecnología (SEP-CONACyT-Mexico
Publisher
Elsevier BV
Subject
Pharmacology,Toxicology
Reference53 articles.
1. Neonatal bisphenol-A exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons;Adewale;Biol. Reprod.,2009
2. The aryl hydrocarbon receptor is required for normal gonadotropin responsiveness in the mouse ovary;Barnett;Toxicol. Appl. Pharmacol.,2007
3. Aryl hydrocarbon receptor regulates growth, but not atresia, of mouse preantral and antral follicles;Benedict;Biol. Reprod.,2003
4. Bisphenol A exposure reduces the estradiol response to gonadotropin stimulation during in vitro fertilization;Bloom;Fertil. Steril.,2011
5. Perinatal exposure to environmentally relevant levels of bisphenol-a decreases fertility and fecundity in CD-1 mice;Cabaton;Environ. Health Perspect.,2011
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