A novel histone deacetylase inhibitor Se-SAHA attenuates isoproterenol-induced heart failure via antioxidative stress and autophagy inhibition-Reference-Cited by-同舟云学术

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A novel histone deacetylase inhibitor Se-SAHA attenuates isoproterenol-induced heart failure via antioxidative stress and autophagy inhibition

Published:2024-06 Issue: Volume:487 Page:116957
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Cheng Tianwei,Liu Chang,Wang Yufei,Li Guangru,Feng Lifeng,Zhang Shengzheng,Qi Bing,Cui Jianlin,Guo Lihong,Cao Lei,Wang Yanming,Qi Zhi,Yang Liang

Funder

Natural Science Foundation of Tianjin Municipality

National Natural Science Foundation of China

Fundamental Research Funds for the Central Universities

Publisher

Elsevier BV

Reference56 articles.

1. Molecular regulation of cardiac hypertrophy;Barry;Int. J. Biochem. Cell Biol.,2008

2. Vorinostat-an overview;Bubna;Indian J. Dermatol.,2015

3. Increased phosphorylation-dependent nuclear export of class II histone deacetylases in failing human heart;Calalb;Clin. Transl. Sci.,2009

4. Histone deacetylase (HDAC) inhibitors attenuate cardiac hypertrophy by suppressing autophagy;Cao;Proc. Natl. Acad. Sci. USA,2011

5. HDAC11 regulates type I interferon signaling through defatty-acylation of SHMT2;Cao;Proc. Natl. Acad. Sci. USA,2019

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