Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration-Reference-Cited by-同舟云学术

Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration

Published:2007-03 Issue:1 Volume:204 Page:220-233
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Bye Nicole,Habgood Mark D.,Callaway Jennifer K.,Malakooti Nakisa,Potter Ann,Kossmann Thomas,Morganti-Kossmann M. Cristina

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference53 articles.

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3. Systemically administered interleukin-10 reduces tumor necrosis factor-alpha production and significantly improves functional recovery following traumatic spinal cord injury in rats;Bethea;J. Neurotrauma,1999

4. Role of IL-1alpha and IL-1beta in ischemic brain damage;Boutin;J. Neurosci.,2001

5. An experimental model of closed head injury in mice: pathophysiology, histopathology, and cognitive deficits;Chen;J. Neurotrauma,1996

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