Relationship between neuropathology and disease progression in the SOD1G93A ALS mouse-Reference-Cited by-同舟云学术

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Relationship between neuropathology and disease progression in the SOD1G93A ALS mouse

Published:2011-02 Issue:2 Volume:227 Page:287-295
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Yang Wendy W.,Sidman Richard L.,Taksir Tatyana V.,Treleaven Christopher M.,Fidler Jonathan A.,Cheng Seng H.,Dodge James C.,Shihabuddin Lamya S.

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference68 articles.

1. Induction of nitrotyrosine-like immunoreactivity in the lower motor neuron of amyotrophic lateral sclerosis;Abe;Neurosci. Lett.,1995

2. Immune reactivity in a mouse model of familial ALS correlates with disease progression;Alexianu;Neurology,2001

3. Development of a rat model of amyotrophic lateral sclerosis expressing a human SOD1 transgene;Aoki;Neuropathology,2005

4. Modulation of intracellular formation of reactive oxygen intermediates in peritoneal macrophages and microglia/brain macrophages by propentofylline;Banati;J. Cereb. Blood Flow Metab.,1994

5. Increased 3-nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis;Beal;Ann. Neurol.,1997

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