IKBKG (nuclear factor-κB essential modulator) mutation can be associated with opportunistic infection without impairing Toll-like receptor function-Reference-Cited by-同舟云学术

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IKBKG (nuclear factor-κB essential modulator) mutation can be associated with opportunistic infection without impairing Toll-like receptor function

Published:2008-04 Issue:4 Volume:121 Page:976-982
ISSN:0091-6749
Container-title:Journal of Allergy and Clinical Immunology
language:en
Short-container-title:Journal of Allergy and Clinical Immunology

Author:

Salt Bryn H.,Niemela Julie E.,Pandey Rahul,Hanson Eric P.,Deering Raquel P.,Quinones Ralph,Jain Ashish,Orange Jordan S.,Gelfand Erwin W.

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference30 articles.

1. A novel X-linked disorder of immune deficiency and hypohidrotic ectodermal dysplasia is allelic to incontinentia pigmenti and due to mutations in IKK-gamma (NEMO);Zonana;Am J Hum Genet,2000

2. The NF-kappaB signaling pathway in human diseases: from incontinentia pigmenti to ectodermal dysplasias and immune-deficiency syndromes;Smahi;Hum Mol Genet,2002

3. Human disease resulting from gene mutations that interfere with appropriate nuclear factor-kappaB activation;Orange;Immunol Rev,2005

4. The presentation and natural history of immunodeficiency caused by nuclear factor kappa B essential modulator mutation;Orange;J Allergy Clin Immunol,2004

5. Human nuclear factor kappa B essential modulator mutation can result in immunodeficiency without ectodermal dysplasia;Orange;J Allergy Clin Immunol,2004

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