C1 inhibitor and prolylcarboxypeptidase modulate prekallikrein activation on endothelial cells-Reference-Cited by-同舟云学术

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C1 inhibitor and prolylcarboxypeptidase modulate prekallikrein activation on endothelial cells

Published:2023-10 Issue:4 Volume:152 Page:961-971.e7
ISSN:0091-6749
Container-title:Journal of Allergy and Clinical Immunology
language:en
Short-container-title:Journal of Allergy and Clinical Immunology

Author:

Merkulova Alona A.,Abdalian Sarah,Silbak Sadiq,Pinheiro Alessandro,Schmaier Alvin H.

Funder

NIH

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference52 articles.

1. The contact activation and kallikrein/kinin systems: pathophysiologic and physiologic activities;Schmaier;J Thromb Haemost,2016

2. Proteolytic properties of single-chain factor XII: a mechanism for triggering contact activation;Ivanov;Blood,2017

3. Kinin release from high molecular weight kininogen by the action of Hageman factor in the absence of kallikrein;Wiggins;J Biol Chem,1983

4. The kinetics of the release of bradykinin by kallikrein in normal human plasma;Girey;J Lab Clin Med,1972

5. Heat shock protein 90 catalyzes activation of the prekallikrein-kininogen complex in the absence of factor XII;Joseph;Proc Natl Acad Sci U S A,2002

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Increased Prolylcarboxypeptidase Expression Can Serve as a Biomarker of Senescence in Culture;Molecules;2024-05-09

2. Is there a role for bradykinin in cerebral malaria pathogenesis?;Frontiers in Cellular and Infection Microbiology;2023-08-10

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