The human complement receptor type 2 (CR2)/CR1 fusion protein TT32, a novel targeted inhibitor of the classical and alternative pathway C3 convertases, prevents arthritis in active immunization and passive transfer mouse models-Reference-Cited by-同舟云学术

The human complement receptor type 2 (CR2)/CR1 fusion protein TT32, a novel targeted inhibitor of the classical and alternative pathway C3 convertases, prevents arthritis in active immunization and passive transfer mouse models

Published:2019-01 Issue: Volume:105 Page:150-164
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Fridkis-Hareli Masha,Storek Michael,Or Eran,Altman Richard,Katti Suresh,Sun Fang,Peng Tao,Hunter Jeff,Johnson Krista,Wang Yi,Lundberg Ante S.,Mehta Gaurav,Banda Nirmal K.,Michael Holers V.

Funder

Alexion Pharmaceuticals

National Institutes of Health

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference47 articles.

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2. Alternative complement pathway activation is essential for inflammation and joint destruction in the passive transfer model of collagen-induced arthritis;Banda;J. Immunol.,2006

3. Targeted inhibition of the complement alternative pathway with complement receptor 2 and factor H attenuates collagen antibody-induced arthritis in mice;Banda;J. Immunol.,2009

4. Complement activation pathways in murine immune complex-induced arthritis and in C3a and C5a generation in vitro;Banda;Clin. Exp. Immunol.,2010

5. Role of C3a receptors, C5a receptors, and complement protein C6 deficiency in collagen antibody-induced arthritis in mice;Banda;J. Immunol.,2012

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