Co-ordinated expression of innate immune molecules during mouse neurulation-Reference-Cited by-同舟云学术

Co-ordinated expression of innate immune molecules during mouse neurulation

Published:2015-12 Issue:2 Volume:68 Page:253-260
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Jeanes Angela,Coulthard Liam G,Mantovani Susanna,Markham Kathryn,Woodruff Trent M^ORCID

Funder

National Health and Medical Research Council of Australia

Australian Research Council Future Fellowship

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference38 articles.

1. Toll-like receptors in ischemia-reperfusion injury;Arumugam;Shock,2009

2. Characterization of C3a and C5a receptors in rat cerebellar granule neurons during maturation. Neuroprotective effect of C5a against apoptotic cell death;Bénard;J. Biol. Chem.,2004

3. Hex homeobox gene controls the transition of the endoderm to a pseudostratified, cell emergent epithelium for liver bud development;Bort;Dev. Biol.,2006

4. Hormonal and embryonic regulation of chemokines IL-, MCP-1 and RANTES in the human endometrium during the window of implantation;Caballero-Campo;Mol. Hum.,2002

5. Complement fragment C3a controls mutual attraction during collective migration;Carmona-Fontaine;Dev. Cell,2011

Cited by 23 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. PARVB deficiency alleviates cisplatin-induced tubular injury by inhibiting TAK1 signaling;Cellular and Molecular Life Sciences;2024-09-05

2. C5aR1 antagonism suppresses inflammatory glial responses and alters cellular signaling in an Alzheimer’s disease mouse model;Nature Communications;2024-08-15

3. The complement system in neurodegenerative and inflammatory diseases of the central nervous system;Frontiers in Neurology;2024-07-03

4. Biallelic variants in CSMD1 are implicated in a neurodevelopmental disorder with intellectual disability and variable cortical malformations;Cell Death & Disease;2024-05-30

5. C5aR1 antagonism suppresses inflammatory glial gene expression and alters cellular signaling in an aggressive Alzheimer’s model;2023-08-22