Dust mite-derived Der f 3 activates a pro-inflammatory program in airway epithelial cells via PAR-1 and PAR-2-Reference-Cited by-同舟云学术

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Dust mite-derived Der f 3 activates a pro-inflammatory program in airway epithelial cells via PAR-1 and PAR-2

Published:2019-05 Issue: Volume:109 Page:1-11
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Li Bizhou,Zou Zehong,Meng Fanmei,Raz Eyal,Huang Yuye,Tao Ailin,Ai Yuncan

Funder

Major Program of National Science and Technology of China

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference68 articles.

1. The house dust mite allergen Der p 1, unlike Der p 3, stimulates the expression of interleukin-8 in human airway epithelial cells via a proteinase-activated receptor-2-independent mechanism;Adam;J. Biol. Chem.,2006

2. Activation of protease-activated receptor (PAR)-1, PAR-2, and PAR-4 stimulates IL-6, IL-8, and prostaglandin E2 release from human respiratory epithelial cells;Asokananthan;J. Immunol.,2002

3. Absence of proteinase-activated receptor-1 signaling in mice confers protection from fMLP-induced goblet cell metaplasia;Atzori;Am. J. Respir. Cell Mol. Biol.,2009

4. Cloning of a group 3 allergen from Blomia tropicalis mites;Cheong;Allergy,2003

5. Mold allergen, pen C 13, induces IL-8 expression in human airway epithelial cells by activating protease-activated receptor 1 and 2;Chiu;J. Immunol.,2007

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