Hyperandrogenemia is implicated in both the metabolic and reproductive morbidities of polycystic ovary syndrome
Author:
Funder
Ewha Womans University
Publisher
Elsevier BV
Subject
Obstetrics and Gynaecology,Reproductive Medicine
Reference24 articles.
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2. Revised 2003 consensus on diagnostic criteria and long-term health risks related to polycystic ovary syndrome;Rotterdam ESHRE/ASRAM-Sponsored PCOS Consensus Workshop Group;Fertil Steril,2004
3. Positions statement: criteria for defining polycystic ovary syndrome as a predominantly hyperandrogenic syndrome: an Androgen Excess Society guideline;Azziz;J Clin Endocrinol Metab,2006
4. Detecting insulin resistance in polycystic ovary syndrome: purposes and pitfalls;Legro;Obstet Gynecol Surv,2004
5. An epidemiological test of the hyperinsulinemia-hypertension hypothesis;Muller;J Clin Endocrinol Metab,1993
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1. Modern view on the etiology and pathogenesis of hyperandrogenic conditions in women of reproductive age against the background of normal levels of male steroid hormones;The Ukrainian Scientific Medical Youth Journal;2024-06-27
2. Long noncoding RNA C-Terminal binding protein 1 antisense regulates ovarian granulosa cells proliferation and autophagy and participates in polycystic ovary syndrome;2023-12-19
3. Identification of Hub Genes and Biomarkers between Hyperandrogen and Normoandrogen Polycystic Ovary Syndrome by Bioinformatics Analysis;Combinatorial Chemistry & High Throughput Screening;2023-01
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