Molecular dynamics investigation on the Asciminib resistance mechanism of I502L and V468F mutations in BCR-ABL-Reference-Cited by-同舟云学术

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Molecular dynamics investigation on the Asciminib resistance mechanism of I502L and V468F mutations in BCR-ABL

Published:2019-06 Issue: Volume:89 Page:242-249
ISSN:1093-3263
Container-title:Journal of Molecular Graphics and Modelling
language:en
Short-container-title:Journal of Molecular Graphics and Modelling

Author:

Zhan Jiu-Yu,Ma Jie,Zheng Qing-Chuan

Funder

Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Materials Chemistry,Computer Graphics and Computer-Aided Design,Physical and Theoretical Chemistry,Spectroscopy

Reference51 articles.

1. Chronic myeloid leukemia;Sawyers;N. Engl. J. Med.,1999

2. Direct quantification of trace amounts of a chronic myeloid leukemia biomarker using locked nucleic acid capture probes;Mishra;Anal. Chem.,2018

3. BCR-ABL1 kinase domain mutations: methodology and clinical evaluation;Alikian;Am. J. Hematol.,2012

4. Philadelphia chromosomepositive leukemias: from basic mechanisms to molecular therapeutics;Kurzrock;Ann. Intern. Med.,2003

5. Expression of a distinctive BCR-ABL oncogene in Ph1-positive acute lymphocytic leukemia (ALL);Clark;Science,1988

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