The flavonoid baicalein rescues synaptic plasticity and memory deficits in a mouse model of Alzheimer’s disease-Reference-Cited by-同舟云学术

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The flavonoid baicalein rescues synaptic plasticity and memory deficits in a mouse model of Alzheimer’s disease

Published:2016-09 Issue: Volume:311 Page:309-321
ISSN:0166-4328
Container-title:Behavioural Brain Research
language:en
Short-container-title:Behavioural Brain Research

Author:

Gu Xun-Hu,Xu Li-Jun,Liu Zhi-Qiang,Wei Bo,Yang Yuan-Jian,Xu Guo-Gang,Yin Xiao-Ping,Wang Wei

Funder

National Natural Science Funds of China

Natural Science Foundation of Jiangxi, China

Publisher

Elsevier BV

Subject

Behavioral Neuroscience

Reference64 articles.

1. Alzheimer's disease is a synaptic failure;Selkoe;Science (New York, N.Y.),2002

2. Grape juice, berries, and walnuts affect brain aging and behavior;Joseph;J. Nutr.,2009

3. Long-term green tea catechin administration prevents spatial learning and memory impairment in senescence-accelerated mouse prone-8 mice by decreasing Abeta1-42 oligomers and upregulating synaptic plasticity-related proteins in the hippocampus;Li;Neuroscience,2009

4. Post-injury baicalein improves histological and functional outcomes and reduces inflammatory cytokines after experimental traumatic brain injury;Chen;Br. J. Pharmacol.,2008

5. Baicalein protects cortical neurons from beta-amyloid (25–35) induced toxicity;Lebeau;Neuroreport,2001

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