The cardiovascular pharmacology of prostacyclin (PGI2) in the rat
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Biochemistry
Reference30 articles.
1. An Enzyme Isolated from Arteries Transforms Prostaglandin Endoperoxides to an Unstable Substance that Inhibits Platelet Aggregation;Moncada;Nature,1976
2. The Chemical Structure of Prostaglandin X (Prostacyclin);Johnson;Prostaglandins,1976
3. Human Arterial and Venous Tissues Generate Prostacyclin (Prostaglandin X) a Potent Inhibitor of Platelet Aggregation;Moncada;Lancet,1977
4. A Lipid Peroxide Inhibits the Enzyme in Blood Vessel Microsomes that Generate from Prostaglandin Endoperoxides the Substance (Prostaglandin X) which Prevents Platelet Aggregation;Moncada;Prostaglandins,1976
5. Lungs as a Generator of Prostacyclin — Hypothesis on Physiological Significance;Gryglewski;Naunyn-Schmiedeberg's Arch. Pharmacol.,1978
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1. Effects of a PPAR?? Agonist, GI262570, on Renal Filtration Fraction and Nitric Oxide Level in Conscious Rats;Journal of Cardiovascular Pharmacology;2003-09
2. Effects of intravenous administration of prostacyclin on regional blood circulation in awake rats;British Journal of Pharmacology;1999-03
3. Systemic and pulmonary hypertension after abrupt cessation of prostacyclin: role of thromboxane A2;Journal of Applied Physiology;1996-01-01
4. Intravenous epoprostenol sodium does not increase hepatic microsomal enzyme activity in rats;Prostaglandins;1995-11
5. Effect of contrast media on in vitro bleeding time: Assessment by a hollow fiber instrument;Academic Radiology;1995-03
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