Transcriptional changes in OXPHOS complex I deficiency are related to anti-oxidant pathways and could explain the disturbed calcium homeostasis-Reference-Cited by-同舟云学术

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Transcriptional changes in OXPHOS complex I deficiency are related to anti-oxidant pathways and could explain the disturbed calcium homeostasis

Published:2012-07 Issue:7 Volume:1822 Page:1161-1168
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Voets A.M.,Huigsloot M.,Lindsey P.J.,Leenders A.M.,Koopman W.J.H.,Willems P.H.G.M.,Rodenburg R.J.,Smeitink J.A.M.,Smeets H.J.M.

Funder

Dutch IOP Genomics

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference54 articles.

1. The genetics and pathology of oxidative phosphorylation;Smeitink;Nat. Rev. Genet.,2001

2. Isolated complex I deficiency in children: clinical, biochemical and genetic aspects;Loeffen;Hum. Mutat.,2000

3. NADH-coenzyme Q reductase (complex I) deficiency: heterogeneity in phenotype and biochemical findings;Pitkanen;J. Inherit. Metab. Dis.,1996

4. Mitochondrial complex I deficiency: from organelle dysfunction to clinical disease;Distelmaier;Brain,2009

5. Human CIA30 is involved in the early assembly of mitochondrial complex I and mutations in its gene cause disease;Dunning;EMBO J.,2007

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