Inhibition of the proteasome preserves Mitofusin-2 and mitochondrial integrity, protecting cardiomyocytes during ischemia-reperfusion injury

Author:

Olmedo Ivonne,Pino Gonzalo,Riquelme Jaime A.,Aranguiz Pablo,Díaz Magda C.,López-Crisosto Camila,Lavandero Sergio,Donoso Paulina,Pedrozo Zully,Sánchez Gina

Funder

Fondo Nacional de Desarrollo Científico y Tecnológico

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference72 articles.

1. Ischemia and reperfusion-from mechanism to translation;Eltzschig;Nat. Med.,2011

2. Myocardial ischemia-reperfusion injury: a neglected therapeutic target;Hausenloy;J. Clin. Invest.,2013

3. ROS and redox signaling in myocardial ischemia-reperfusion injury and cardioprotection;Cadenas;Free Radic. Biol. Med.,2018

4. Dong Y, Undyala V, Przyklenk K: Inhibition of mitochondrial fission as a molecular target for cardioprotection: critical importance of the timing of treatment. Basic Res. Cardiol. 2016;111:1–17.

5. Changes in mitochondrial dynamics during ceramide-induced cardiomyocyte early apoptosis;Parra;Cardiovasc. Res.,2008

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