Submicromolar Aβ42 reduces hippocampal glutamate receptors and presynaptic markers in an aggregation-dependent manner-Reference-Cited by-同舟云学术

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Submicromolar Aβ42 reduces hippocampal glutamate receptors and presynaptic markers in an aggregation-dependent manner

Published:2011-12 Issue:12 Volume:1812 Page:1664-1674
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Wisniewski Meagan L.,Hwang Jeannie,Bahr Ben A.

Funder

National Institutes of Health

Merz Pharmaceuticals GmbH

North Carolina Biotechnology Center

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference66 articles.

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2. Aβ oligomers – a decade of discovery;Walsh;J. Neurochem.,2007

3. The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics;Hardy;Science,2002

4. Water-soluble Aβ (N-40, N-42) oligomers in normal and Alzheimer disease brains;Kuo;J. Biol. Chem.,1996

5. Assembly and aggregation properties of synthetic Alzheimer's A4/β amyloid peptide analogs;Burdick;J. Biol. Chem.,1992

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