IGF2 derived from SH-SY5Y neuroblastoma cells induces the osteoclastogenesis of human monocytic precursors-Reference-Cited by-同舟云学术

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IGF2 derived from SH-SY5Y neuroblastoma cells induces the osteoclastogenesis of human monocytic precursors

Published:2011-09 Issue:15 Volume:317 Page:2147-2158
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Avnet Sofia,Salerno Manuela,Quacquaruccio Gianni,Granchi Donatella,Giunti Armando,Baldini Nicola

Funder

Associazione Italiana per la Ricerca sul Cancro

Ministero della Salute

Publisher

Elsevier BV

Subject

Cell Biology

Reference52 articles.

1. Mechanisms of bone metastasis;Roodman;N. Engl. J. Med.,2004

2. Clinical features of metastatic bone disease and risk of skeletal morbidity;Coleman;Clin. Cancer Res.,2006

3. Therapeutic relevance of osteoprotegerin gene therapy in osteosarcoma: blockade of the vicious cycle between tumor cell proliferation and bone resorption;Lamoureux;Cancer Res.,2007

4. Basic mechanisms responsible for osteolytic and osteoblastic bone metastases;Guise;Clin. Cancer Res.,2006

5. Lytic bone lesions in human neuroblastoma xenograft involve osteoclast recruitment and are inhibited by bisphosphonate;Sohara;Cancer Res.,2003

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Involvement of Noncoding RNAs in the Differentiation of Osteoclasts;Stem Cells International;2020-08-25

2. Preclinical models for neuroblastoma: Advances and challenges;Cancer Letters;2020-04

3. Histochemical evidence of IGF2 mRNA-binding protein 2-mediated regulation of osteoclast function and adhesive ability;Histochemistry and Cell Biology;2018-01-10

4. MDA-MB-231 breast cancer cells fuel osteoclast metabolism and activity: A new rationale for the pathogenesis of osteolytic bone metastases;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2017-12

5. Potassium citrate prevents increased osteoclastogenesis resulting from acidic conditions: Implication for the treatment of postmenopausal bone loss;PLOS ONE;2017-07-17

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