Inducible Expression of a p56Lck Transgene Reveals a Central Role for Lck in the Differentiation of CD4 SP Thymocytes-Reference-Cited by-同舟云学术

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Inducible Expression of a p56Lck Transgene Reveals a Central Role for Lck in the Differentiation of CD4 SP Thymocytes

Published:2000-05 Issue:5 Volume:12 Page:537-546
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Legname Giuseppe,Seddon Ben,Lovatt Matthew,Tomlinson Peter,Sarner Nitza,Tolaini Mauro,Williams Keith,Norton Trisha,Kioussis Dimitris,Zamoyska Rose

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference36 articles.

1. Inhibition of T-cell receptor β-chain gene rearrangement by overexpression of the non-receptor protein tyrosine kinase p56 lck;Anderson;EMBO J.,1992

2. Protein tyrosine kinase p56lck controls allelic exclusion of T cell receptor β-chain genes;Anderson;Nature,1993

3. CD3 ligation on immature thymocytes generates antagonist-like signals appropriate for CD8 lineage commitment, independently of T cell receptor specificity;Basson;J. Exp. Med.,1998

4. Molecular requirements for lineage commitment in the thymus—antibody-mediated receptor engagements reveal a central role for lck in lineage decisions;Basson;Immunol. Rev.,1998

5. Signals through CD8 or CD4 can induce commitment to the CD4 lineage in the thymus;Bommhardt;Eur. J. Immunol.,1997

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