Intracellular trafficking in Plasmodium-infected erythrocytes
Author:
Publisher
Elsevier BV
Subject
Infectious Diseases,Microbiology (medical),Microbiology
Reference44 articles.
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1. Maurer's clefts, the enigma of Plasmodium falciparum;Proceedings of the National Academy of Sciences;2013-11-27
2. The N-terminal segment of Plasmodium falciparum SURFIN4.1 is required for its trafficking to the red blood cell cytosol through the endoplasmic reticulum;Parasitology International;2013-04
3. Cytoplasmic free Ca2+ is essential for multiple steps in malaria parasite egress from infected erythrocytes;Malaria Journal;2013-01-30
4. The Potent Antimalarial Drug Cyclosporin A Preferentially Destabilizes Sphingomyelin-Rich Membranes;Langmuir;2009-08-21
5. Proteins of the Plasmodium falciparum two transmembrane Maurer’s cleft protein family, PfMC-2TM, and the 130 kDa Maurer’s cleft protein define different domains of the infected erythrocyte intramembranous network;Parasitology Research;2009-01-07
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