IL-2, IL-4, IFN-γ or TNF-α enhances BAFF-stimulated cell viability and survival by activating Erk1/2 and S6K1 pathways in neoplastic B-lymphoid cells-Reference-Cited by-同舟云学术

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IL-2, IL-4, IFN-γ or TNF-α enhances BAFF-stimulated cell viability and survival by activating Erk1/2 and S6K1 pathways in neoplastic B-lymphoid cells

Published:2016-08 Issue: Volume:84 Page:37-46
ISSN:1043-4666
Container-title:Cytokine
language:en
Short-container-title:Cytokine

Author:

Gui Lin,Zeng Qingyu,Xu Zhigang,Zhang Hai,Qin Shanshan,Liu Chunxiao,Xu Chong,Qian Zhou,Zhang Shuangquan,Huang Shile^ORCID,Chen Long

Funder

National Natural Science Foundation of China

NIH

Project for the Priority Academic Program Development of Jiangsu Higher Education Institutions of China

American Cancer Society

Louisiana Board of Regents

Innovative Research Program of Jiangsu College Graduate of China

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference52 articles.

1. B-cell responses to B-cell activation factor of the TNF family (BAFF) are impaired in the absence of PI3K delta;Henley;Eur. J. Immunol.,2008

2. Critical role of monocytes to support normal B cell and diffuse large B cell lymphoma survival and proliferation;Mueller;J. Leukoc. Biol.,2007

3. BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growth;Schneider;J. Exp. Med.,1999

4. BLyS: member of the tumor necrosis factor family and B lymphocyte stimulator;Moore;Science,1999

5. TALL-1 is a novel member of the TNF family that is down-regulated by mitogens;Shu;J. Leukoc. Biol.,1999

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