DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination-Reference-Cited by-同舟云学术

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DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination

Published:2017-02 Issue:4 Volume:65 Page:671-684.e5
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Biehs Ronja,Steinlage Monika,Barton Olivia,Juhász Szilvia,Künzel Julia,Spies Julian,Shibata Atsushi,Jeggo Penny A.,Löbrich Markus

Funder

Deutsche Forschungsgemeinschaft

European Union

Bundesministerium für Bildung und Forschung

Medical Research Council

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference47 articles.

1. Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1;Barton;J. Cell Biol.,2014

2. ATM and Artemis promote homologous recombination of radiation-induced DNA double-strand breaks in G2;Beucher;EMBO J.,2009

3. EXD2 promotes homologous recombination by facilitating DNA end resection;Broderick;Nat. Cell Biol.,2016

4. Unifying the DNA end-processing roles of the artemis nuclease: Ku-dependent artemis resection at blunt DNA ends;Chang;J. Biol. Chem.,2015

5. Coordinated nuclease activities counteract Ku at single-ended DNA double-strand breaks;Chanut;Nat. Commun.,2016

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