Rad5 Recruits Error-Prone DNA Polymerases for Mutagenic Repair of ssDNA Gaps on Undamaged Templates-Reference-Cited by-同舟云学术

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Rad5 Recruits Error-Prone DNA Polymerases for Mutagenic Repair of ssDNA Gaps on Undamaged Templates

Published:2019-03 Issue:5 Volume:73 Page:900-914.e9
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Gallo David,Kim TaeHyung,Szakal Barnabas,Saayman Xanita,Narula Ashrut,Park Yoona,Branzei Dana,Zhang Zhaolei,Brown Grant W.

Funder

Canadian Cancer Society

Natural Sciences and Engineering Research Council of Canada

Ontario Graduate Scholarship

Italian Association for Cancer Research

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference75 articles.

1. Unprotected replication forks are converted into mitotic sister chromatid bridges;Ait Saada;Mol. Cell,2017

2. Assembly of Slx4 signaling complexes behind DNA replication forks;Balint;EMBO J.,2015

3. Mcm10 deficiency causes defective-replisome-induced mutagenesis and a dependency on error-free postreplicative repair;Becker;Cell Cycle,2014

4. Mutations in replicative stress response pathways are associated with S phase-specific defects in nucleotide excision repair;Bélanger;J. Biol. Chem.,2016

5. Rev3, the catalytic subunit of Polζ, is required for maintaining fragile site stability in human cells;Bhat;Nucleic Acids Res.,2013

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1. The cytidine deaminase APOBEC3C has unique sequence and genome feature preferences;GENETICS;2024-07-01

2. Rad51 determines pathway usage in post-replication repair;2024-06-14

3. EXO1 and DNA2-mediated ssDNA gap expansion is essential for ATR activation and to maintain viability in BRCA1-deficient cells;Nucleic Acids Research;2024-05-09

4. EXO1-mediated ssDNA gap expansion is essential for ATR activation and to maintain viability in BRCA1-deficient cells;2024-01-23

5. Strand asymmetry of DNA damage tolerance mechanisms;2024-01-22

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