m6A Modification Prevents Formation of Endogenous Double-Stranded RNAs and Deleterious Innate Immune Responses during Hematopoietic Development

Author:

Gao Yimeng,Vasic Radovan,Song Yuanbin,Teng Rhea,Liu Chengyang,Gbyli Rana,Biancon Giulia,Nelakanti Raman,Lobben Kirsten,Kudo Eriko,Liu Wei,Ardasheva Anastasia,Fu Xiaoying,Wang Xiaman,Joshi Poorval,Lee Veronica,Dura Burak,Viero Gabriella,Iwasaki Akiko,Fan Rong,Xiao Andrew,Flavell Richard A.,Li Hua-Bing,Tebaldi Toma,Halene StephanieORCID

Funder

State of Connecticut

NIH

Frederick A. Deluca Foundation

National Natural Science Foundation of China

Shanghai Institutions of Higher Learning

Howard Hughes Medical Institute

James Hudson Brown-Alexander Brown Coxe Postdoctoral Fellowships

American Society of Hematology

William U. Gardner Memorial Student Research Fellowship

Yale Cooperative Center of Excellence in Hematology

CNR Short Term Mobility 2018

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference66 articles.

1. Nucleoside modifications in RNA limit activation of 2′-5′-oligoadenylate synthetase and increase resistance to cleavage by RNase L;Anderson;Nucleic Acids Res.,2011

2. Promoter-bound METTL3 maintains myeloid leukaemia by m6A-dependent translation control;Barbieri;Nature,2017

3. m(6)A RNA modification controls cell fate transition in mammalian embryonic stem cells;Batista;Cell Stem Cell,2014

4. The SMAD2/3 interactome reveals that TGFβ controls m6A mRNA methylation in pluripotency;Bertero;Nature,2018

5. Proapoptotic signaling induced by RIG-I and MDA-5 results in type I interferon-independent apoptosis in human melanoma cells;Besch;J. Clin. Invest.,2009

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