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Gon4l/Udu regulates cardiomyocyte proliferation and maintenance of ventricular chamber identity during zebrafish development

  • Published:2020-06 Issue:2 Volume:462 Page:223-234
  • ISSN:0012-1606
  • Container-title:Developmental Biology
  • language:en
  • Short-container-title:Developmental Biology

Author:

Budine Terin E.,de Sena-Tomás Carmen,Williams Margot L.K.,Sepich Diane S.,Targoff Kimara L.,Solnica-Krezel Lila

Funder

National Institute of General Medical Sciences

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference78 articles.

1. Screeening mosaic F1 females for mutations affecting zebrafish heart induction and patterning;Alexander,1998

2. Zebrafish as a model to study cardiac development and human cardiac disease;Bakkers;Cardiovasc. Res.,2011

3. Early B cell progenitors deficient for GON4L fail to differentiate due to a block in mitotic cell division;Barr;J. Immunol.,2017

4. Proliferation of embryonic cardiomyocytes in zebrafish requires the sodium channel scn5Lab;Bennett;Genesis,2013

5. tp53 mutant zebrafish develop malignant peripheral nerve sheath tumors;Berghmans;Proc. Natl. Acad. Sci. U. S. A.,2005
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