Evidence of functional redundancy between MID proteins: implications for the presentation of Opitz syndrome
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Developmental Biology,Molecular Biology
Reference32 articles.
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3. Functional characterization of the Opitz syndrome gene product (midin): evidence for homodimerization and association with microtubules throughout the cell cycle;Cainarca;Hum. Mol. Genet.,1999
4. Mechanisms of left–right determination in vertebrates;Capdevila;Cell,2000
5. Improved method for chick whole-embryo culture using a filter paper carrier;Chapman;Dev. Dyn.,2001
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1. Morphometric analysis of the size-adjusted linear dimensions of the skull landmarks revealed craniofacial dysmorphology in Mid1-cKO mice;BMC Genomics;2023-02-09
2. MID1 and MID2 regulate cell migration and epithelial-mesenchymal transition via modulating Wnt/β-catenin signaling;Annals of Translational Medicine;2020-08
3. The MID1 gene product in physiology and disease;Gene;2020-07
4. Emerging Roles of the TRIM E3 Ubiquitin Ligases MID1 and MID2 in Cytokinesis;Frontiers in Physiology;2019-03-19
5. Mid1/Mid2 expression in craniofacial development and a literature review of X‐linked Opitz syndrome;Molecular Genetics & Genomic Medicine;2015-12-12
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