Liver steatosis, cardiac and renal fibrosis, and hypertension in overweight rats: Angiotensin-(3–4)-sensitive hepatocardiorenal syndrome

Author:

Crisóstomo Thuany,Pardal Marco A.E.,Herdy Simone A.,Muzi-Filho Humberto,Mello Debora B.,Takiya Christina M.,Luzes Rafael,Vieyra AdalbertoORCID

Funder

FAPERJ

CAPES

CNPq

Publisher

Elsevier BV

Subject

General Medicine

Reference22 articles.

1. Angiotensin-(3–4) normalizes blood pressure, decreases Na+ and energy intake, but preserves urinary Na+ excretion in overweight hypertensive rats;Luzes;Biochim Biophys Acta (BBA) - Mol Basis Dis,2021

2. How western diet and lifestyle drive the pandemic of obesity and civilization diseases;Kopp;Diabetes Metab Syndr Obes,2019

3. Is angiotensin-(3–4) (Val-Tyr), the shortest angiotensin II-derived peptide, opening new vistas on the renin-angiotensin system?;Dias;J Renin-Angiotensin-Aldosterone Syst JRAAS,2017

4. Angiotensin-(3–4) counteracts the Angiotensin II inhibitory action on renal Ca2+-ATPase through a cAMP/PKA pathway;Axelband;Regul Pept,2012

5. Cardio-renal syndromes: a systematic approach for consensus definition and classification;Ronco;Heart Fail Rev,2012

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