Inhibition of GABA-stimulated chloride influx by the convulsant benzodiazepines Ro 5-3663 and Ro 5-4864 into membrane vesicles from rat cerebral cortex
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference4 articles.
1. The effect of benzodiazepines and ß-carbolines on GABA-stimulated chloride influx by membrane vesicles from rat cerebral cortex;Obata;Biochem. Biophys. Res. Commun.,1986
2. The inhibition of GABA-stimulated benzodiazepine binding by a convulsant benzodiazepine;O'Brien;Life Sci.,1980
3. Convulsant binding sites on the benzodiazepine/GABA receptor;Ticku,1986
4. Electrophysiological and pharmacological actions of the convulsant benzodiazepine Ro 5-4864;Weissman;European J. Pharmacol.,1984
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1. Anticonvulsant activity of δ-HCH, calcium channel blockers and calmodulin antagonists in seizures induced by lindane and other convulsant drugs;Brain Research;1993-09
2. The GABAA receptor channel mediated chloride ion translocation through the plasma membrane: New insights from36Cl? ion flux measurements;Synapse;1993-01
3. Ro 5-4864, like picrotoxin, enhances epsp-spike coupling in the freely behaving rat;Brain Research Bulletin;1991-07
4. Toxicokinetics of Ro 5-4864, lindane and picrotoxin compared;Pharmacology Biochemistry and Behavior;1991-02
5. From binding studies to the molecular biology of GABA receptors;Neurochemical Research;1990-02
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