Mitochondrial GPX4 acetylation is involved in cadmium-induced renal cell ferroptosis-Reference-Cited by-同舟云学术

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Mitochondrial GPX4 acetylation is involved in cadmium-induced renal cell ferroptosis

Published:2024-07 Issue: Volume:73 Page:103179
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Guo Yue-Yue,Liang Nan-Nan,Zhang Xiao-Yi,Ren Ya-Hui,Wu Wen-Zheng,Liu Zhi-Bing,He Yi-Zhang,Zhang Yi-Hao,Huang Yi-Chao,Zhang Tao,Xu De-Xiang,Xu Shen

Funder

Anhui Medical University

National Natural Science Foundation of China

Natural Science Foundation of Anhui Province

Publisher

Elsevier BV

Reference51 articles.

1. Acute kidney injury in the critically ill: an updated review on pathophysiology and management;Pickkers;Intensive Care Med.,2021

2. Kidney physiology and susceptibility to acute kidney injury: implications for renoprotection;Scholz;Nat. Rev. Nephrol.,2021

3. Acute kidney injury from sepsis: current concepts, epidemiology, pathophysiology, prevention and treatment;Peerapornratana;Kidney Int.,2019

4. Drug-induced acute kidney injury;Perazella;Clin. J. Am. Soc. Nephrol.: CJASN,2022

5. Fluctuating risk of acute kidney injury-related mortality for four weeks after exposure to air pollution: a multi-country time-series study in 6 countries;Min;Environ. Int.,2024

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Plin4 exacerbates cadmium-decreased testosterone level via inducing ferroptosis in testicular Leydig cells;Redox Biology;2024-10

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