Mitohormesis during advanced stages of Duchenne muscular dystrophy reveals a redox-sensitive creatine pathway that can be enhanced by the mitochondrial-targeting peptide SBT-20
Author:
Funder
Canada Research Chairs
Canadian Institutes of Health Research
NSERC
Ontario Research Foundation
Canada Foundation for Innovation
James H. Cummings Foundation
Heart and Stroke Foundation of Canada
Publisher
Elsevier BV
Reference30 articles.
1. The TREAT-NMD DMD Global Database: analysis of more than 7,000 Duchenne muscular dystrophy mutations;Bladen;Hum. Mutat.,2015
2. Absence of dystrophin disrupts skeletal muscle signaling: roles of Ca2+, reactive oxygen species, and nitric oxide in the development of muscular dystrophy;Allen;Physiol. Rev.,2016
3. Standard of care versus new-wave corticosteroids in the treatment of Duchenne muscular dystrophy: can we do better?;Kourakis;Orphanet J. Rare Dis.,2021
4. Cardiac therapies for Duchenne muscular dystrophy;Shah;Ther. Adv. Neurol. Disord.,2023
5. Mitochondrial stress responses in Duchenne muscular dystrophy: metabolic dysfunction or adaptive reprogramming?;Bellissimo;Am. J. Physiol. Cell Physiol.,2022
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