Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis

Author:

de Souza Patrícia Reis,Guimarães Francielle Rodrigues,Sales-Campos HelioswiltonORCID,Bonfá Giuliano,Nardini Viviani,Chica Javier Emilio Lazo,Turato Walter Miguel,Silva João Santana,Zamboni Dario Simões,Cardoso Cristina Ribeiro de BarrosORCID

Funder

Núcleo de Apoio à Pesquisa em Doenças Inflamatórias [NAPDIN]

Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)

Fundação de Amparo à Pesquisa do Estado de Minas Gerais (FAPEMIG)

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Publisher

Elsevier BV

Subject

Hematology,Immunology,Immunology and Allergy

Reference32 articles.

1. Identical T cell clones are located within the mouse gut epithelium and lamina propia and circulate in the thoracic duct lymph;Arstila;J. Exp. Med.,2000

2. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells;Bettelli;Nature,2006

3. Modulation of mucosal immunity in a murine model of food-induced intestinal inflammation;Cardoso;Clin. Exp. Allergy,2008

4. Gene-microbiota interactions contribute to the pathogenesis of inflammatory bowel disease;Chu;Science,2016

5. NOD2-mediated dysbiosis predisposes mice to transmissible colitis and colorectal cancer;Couturier-Maillard;J. Clin. Invest.,2013

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