Acute lipopolysaccharide priming boosts inflammasome activation independently of inflammasome sensor induction-Reference-Cited by-同舟云学术

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Acute lipopolysaccharide priming boosts inflammasome activation independently of inflammasome sensor induction

Published:2012-12 Issue:12 Volume:217 Page:1325-1329
ISSN:0171-2985
Container-title:Immunobiology
language:en
Short-container-title:Immunobiology

Author:

Schroder Kate,Sagulenko Vitaliya,Zamoshnikova Alina,Richards Ayanthi A.,Cridland Jasmyn A.,Irvine Katharine M.,Stacey Katryn J.,Sweet Matthew J.

Funder

Australian National Health and Medical Research Council (NHMRC)

NHMRC C.J. Martin Fellowship

KJS by Australian Research Council (ARC) Future Fellowship

MJS by ARC Future Fellowship

Honorary Australian NHMRC Senior Research Fellowship

Publisher

Elsevier BV

Subject

Hematology,Immunology,Immunology and Allergy

Reference11 articles.

1. Cutting edge: reactive oxygen species inhibitors block priming, but not activation, of the NLRP3 inflammasome;Bauernfeind;J. Immunol.,2011

2. Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression;Bauernfeind;J. Immunol.,2009

3. Inflammasome activators induce interleukin-1alpha secretion via distinct pathways with differential requirement for the protease function of caspase-1;Gross;Immunity,2012

4. Type I interferon inhibits interleukin-1 production and inflammasome activation;Guarda;Immunity,2011

5. Identification of a specific interleukin 1 inhibitor in the urine of febrile patients;Liao;J. Exp. Med.,1984

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