Amitriptyline increases GABA-stimulated 36Cl− influx by recombinant (α1γ2) GABAA receptors-Reference-Cited by-同舟云学术

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Amitriptyline increases GABA-stimulated 36Cl− influx by recombinant (α1γ2) GABAA receptors

Published:1999-12 Issue:1-2 Volume:851 Page:277-280
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Malatynska Ewa,Miller Cortland,Schindler Nancy,Cecil Alicia,Knapp Angela,Crites Glenda,Rogers Howell

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference14 articles.

1. Validation of a therapeutic plasma level range in amitriptyline treatment of depression;Breyer-Pfaff;J. Clin. Psychopharmacol.,1989

2. Diazepam enhancement of GABA-gated currents in binary and ternary GABAA receptors: relationship to benzodiazepine binding site density;Granja;J. Mol. Neurosci.,1997

3. Amoxapine inhibition of GABA-stimulated chloride conductance: investigations of potential sites of activity;Ikeda;Life Sci.,1989

4. From binding studies to the molecular biology of GABA receptors;Knapp;Neurochem. Res.,1990

5. Schild regression analysis of antidepressant and bicuculline antagonist effects at the GABAA receptor;Malatynska;Pharmacology,1998

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3. Role of GABAA Receptors in the Mechanism of In Vivo Psychotropic Activity of Amitriptyline in Rats;Bulletin of Experimental Biology and Medicine;2020-01

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