NOX2-Induced Myocardial Fibrosis and Diastolic Dysfunction
Author:
Funder
U.S. Public Health Service
National Institutes of Health
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference12 articles.
1. The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology;Bedard;Physiol Rev,2007
2. Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system;Lassègue;Circ Res,2012
3. Role of endothelial Nox2 NADPH oxidase in angiotensin II–induced hypertension and vasomotor dysfunction;Murdoch;Basic Res Cardiol,2011
4. NAD(P)H oxidase mediates angiotensin II–induced vascular macrophage infiltration and medial hypertrophy;Liu;Arterioscler Thromb Vasc Biol,2003
5. Pivotal role of a gp91(phox)-containing NADPH oxidase in angiotensin II–induced cardiac hypertrophy in mice;Bendall;Circulation,2002
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