Iron accumulation induces oxidative stress, while depressing inflammatory polarization in human iPSC-derived microglia-Reference-Cited by-同舟云学术

Iron accumulation induces oxidative stress, while depressing inflammatory polarization in human iPSC-derived microglia

Published:2022-06 Issue:6 Volume:17 Page:1351-1365
ISSN:2213-6711
Container-title:Stem Cell Reports
language:en
Short-container-title:Stem Cell Reports

Author:

Kenkhuis Boyd^ORCID,van Eekeren Michelle,Parfitt David A.,Ariyurek Yavuz,Banerjee Poulomi,Priller Josef,van der Weerd Louise,van Roon-Mom Willeke M.C.

Funder

Alzheimer Netherlands

Leids Universitair Medisch Centrum

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Genetics,Biochemistry

Reference46 articles.

1. iPSC-derived human microglia-like cells to study neurological diseases;Abud;Neuron,2017

2. Cell iron status influences macrophage polarization;Agoro;PLoS One,2018

3. Cerebral quantitative susceptibility mapping predicts amyloid-β-related cognitive decline;Ayton;Brain,2017

4. Brain iron is associated with accelerated cognitive decline in people with Alzheimer pathology;Ayton;Mol. Psychiatry,2020

5. Regional brain iron associated with deterioration in Alzheimer’s disease: a large cohort study and theoretical significance;Ayton;Alzheimer’s Dement.,2021

Cited by 29 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Deterioration of neuroimmune homeostasis in Alzheimer’s Disease patients who survive a COVID-19 infection;Journal of Neuroinflammation;2024-08-17

2. Knockdown of microglial iron import gene, DMT1, worsens cognitive function and alters microglial transcriptional landscape in a sex-specific manner in the APP/PS1 model of Alzheimer’s disease;2024-06-27

3. The role of labile iron on brain proteostasis; could it be an early event of neurodegenerative disease?;Archives of Biochemistry and Biophysics;2024-06

4. Inflammation-induced TRPV4 channels exacerbate blood–brain barrier dysfunction in multiple sclerosis;Journal of Neuroinflammation;2024-03-23

5. Parkinson’s disease-derived α-synuclein assemblies combined with chronic-type inflammatory cues promote a neurotoxic microglial phenotype;Journal of Neuroinflammation;2024-02-21