Ca2+ Current and Charge Movements in Skeletal Myotubes Promoted by the β-Subunit of the Dihydropyridine Receptor in the Absence of Ryanodine Receptor Type 1-Reference-Cited by-同舟云学术

Ca2+ Current and Charge Movements in Skeletal Myotubes Promoted by the β-Subunit of the Dihydropyridine Receptor in the Absence of Ryanodine Receptor Type 1

Published:2003-02 Issue:2 Volume:84 Page:942-959
ISSN:0006-3495
Container-title:Biophysical Journal
language:en
Short-container-title:Biophysical Journal

Author:

Ahern Chris A.,Sheridan David. C.,Cheng Weijun,Mortenson Lindsay,Nataraj Priya,Allen Paul,Waard Michel De,Coronado Roberto

Publisher

Elsevier BV

Subject

Biophysics

Reference62 articles.

1. Heterologous expression of BI Ca2+ channels in dysgenic myotubes;Adams;J. Gen. Physiol.,1994

2. Expression of cardiac DHPR α1C in α1−/β1− double mutant skeletal muscle myotubes;Ahern;Biophys. J.,1999

3. Intramembrane charge movements and excitation-contraction coupling expressed by two-domain fragments of the Ca2+ channel;Ahern;Proc. Natl. Acad. Sci. USA.,2001

4. A component of excitation-contraction coupling triggered in the absence of the T671–L690 and L720–Q765 regions of the II–III loop of the dihydropyridine receptor α1S pore subunit;Ahern;Biophys. J.,2001

5. Re-examining the RyR1-to-DHPR retrograde signal: Full Ca2+ current in dyspedic myotubes expressing DHPR β2a;Ahern;Biophys. J.,2002

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