On rat renal aminolevulinate transport and metabolism in experimental Fanconi syndrome
Author:
Publisher
Elsevier BV
Subject
Biochemistry,Endocrinology, Diabetes and Metabolism
Reference24 articles.
1. Hereditary Tyrosinemia and the Heme Biosynthetic Pathway. PROFOUND INHIBITION OF δ-AMINOLEVULINIC ACID DEHYDRATASE ACTIVITY BY SUCCINYLACETONE
2. Type I Tyrosinemia: Lack of Immunologically Detectable Fumarylacetoacetase Enzyme Protein in Tissues and Cell Extracts
3. Inhibition of delta-aminolevulinic acid dehydrase by 4,6-dioxoheptanoic acid.
4. Effect of thyroid deficiency on oxidative phosphorylation in rat liver, kidney, and brain mitochondria
5. Effects of succinylacetone on methyl α-d-glucoside uptake by the rat renal tubule
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Tissue distribution of succinylacetone in the rat in vivo: a possible basis for neurotoxicity in hereditary infantile tyrosinemia;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;1993-10-20
2. Brush border membrane proteins in experimental Fanconi's syndrome induced by 4-pentenoate and maleate;Canadian Journal of Physiology and Pharmacology;1992-09-01
3. δ-aminolevulinic acid dehydratase: Effects of succinylacetone in rat liver and kidney in an in vivo model of the renal Fanconi syndrome;Biochemical Medicine and Metabolic Biology;1992-08
4. Physiological basis for an animal model of the renal Fanconi syndrome: use of succinylacetone in the rat;Clinical Science;1992-07-01
5. Renal heme metabolism in hereditary tyrosinemia: use of succinylacetone in rat renal tubules;Biochimica et Biophysica Acta (BBA) - Biomembranes;1991-12
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