High-threshold calcium channel activity in rat hippocampal neurones during hypoxia1Published on the World Wide Web on 19 May 1999.1

Author:

Shkryl V.M.,Nikolaenko L.M.,Kostyuk P.G.,Lukyanetz E.A.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference33 articles.

1. Causes of calcium accumulation in rat cortical brain slices during hypoxia and ischemia: role of ion channels and membrane damage;Bickler;Brain Res.,1994

2. Glutamate neurotoxicity and diseases of the nervous system;Choi;Neuron,1988

3. Downregulation of tetrodotoxin-resistant sodium currents and upregulation of a rapidly repriming tetrodotoxin-sensitive sodium current in small spinal sensory neurons after nerve injury;Cummins;J. Neurosci.,1997

4. Ionic mechanisms of electrical excitability in rat sensory neurons during postnatal ontogenesis;Fedulova;Neuroscience,1991

5. A. Franco-Obregón, J. Ureňa, T. Smani, J. López-Barneo, Calcium channels, cytosolic calcium, and the vasomotor responses to hypoxia, in: J. López-Barneo, E.K. Weir, (Eds.), Oxygen Regulation of Ion Channels and Gene Expression, Armonk, NY: Futura Publishing Company, Inc., 1998, pp. 255–270.

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