Inhibition of NF-κB activity by BAY 11-7082 increases apoptosis in multidrug resistant leukemic T-cell lines-Reference-Cited by-同舟云学术

Inhibition of NF-κB activity by BAY 11-7082 increases apoptosis in multidrug resistant leukemic T-cell lines

Published:2005-12 Issue:12 Volume:29 Page:1425-1434
ISSN:0145-2126
Container-title:Leukemia Research
language:en
Short-container-title:Leukemia Research

Author:

García Mariana G.,Alaniz Laura,Lopes Eloisi C.,Blanco Guillermo,Hajos Silvia E.,Alvarez Elida

Publisher

Elsevier BV

Subject

Cancer Research,Oncology,Hematology

Reference40 articles.

1. Multidrug resistance in cancer: role of ATP-dependent transporters;Gottesman;Nat Rev Cancer,2002

2. Analysis of random recombination between human MDR1 and mouse mdr1a cDNA in a pHaMDR-dihydrofolate reductase bicistronic expression system;Shoshani;Mol Pharmacol,1998

3. Reversal of cisplatin and multidrug resistance by ribozyme-mediated glutathione suppression;Nagata;Biochem Biophys Res Commun,2001

4. Mechanisms of tolerance to DNA damaging therapeutic drugs;Karran;Carcinogenesis,2001

5. Current status of the molecular mechanisms of anticancer drug-induced apoptosis. The contribution of molecular-level analysis to cancer chemotherapy;Kim;Cancer Chemother Pharmacol,2002

Cited by 84 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. In Vivo Effects of Bay 11-7082 on Fibroid Growth and Gene Expression: A Preclinical Study;Cells;2024-06-24

2. Role of the NLRP3 inflammasome in gynecological disease;Biomedicine & Pharmacotherapy;2023-10

3. Free-fatty acid receptor-1 (FFA1/GPR40) promotes papillary RCC proliferation and tumor growth via Src/PI3K/AKT/NF-κB but suppresses migration by inhibition of EGFR, ERK1/2, STAT3 and EMT;Cancer Cell International;2023-06-24

4. Binding of Gamma-Glutamyl Transferase to TLR4 Signalling Allows Tissue Factor Activation in Monocytes;International Journal of Molecular Sciences;2022-10-13

5. The NLRP3 Inflammasome Pathway: A Review of Mechanisms and Inhibitors for the Treatment of Inflammatory Diseases;Frontiers in Aging Neuroscience;2022-06-10