CML patient with rare b2a3 (e13a3) variant of BCR–ABL transcript: Complete molecular response to imatinib
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Oncology,Hematology
Reference8 articles.
1. CML with an e1a3 BCR–ABL fusion: rare, benign, and a potential diagnostic pitfall;Al-Ali;Blood,2002
2. Chronic myelogonous leukemia with e13a3 (b2a3) type of BCR–ABL having a DNA breakpoint between ABL exons a2 and a3;Liu;Am J Hematol,2003
3. Chronic myeloid leukemia with an e13a3 BCR–ABL fusion: benign course responsive to imatinib with an RT-PCR advisory;Snyder;Am J Hematol,2004
4. A new case of chronic myeloid leukemia (CML) in myeloid blast crisis with an atypical (b3/a3) junction of the BCR/ABL gene;Polák;Leukemia,1998
5. A rare, in frame BCR–ABL fusion (e13a3) in a patient with an aggressive chronic myeloid leukemia;Otazú;Acta Haematol,2002
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1. Chronic myeloid leukemia with two rare fusion gene transcripts of atypical BCR::ABL: A case report and literature review;Medicine;2024-01-19
2. Burden of acute toxicities in head-and-neck radiation therapy: A single-institutional experience;South Asian Journal of Cancer;2019-04
3. Chronic myeloid leukemia with a rare fusion transcript, b2a3 (e13a3) BCR–ABL1: A report of four cases from India;South Asian Journal of Cancer;2019-04
4. A rare e13a3 (b2a3) BCR-ABL1 fusion transcript with normal karyotype in chronic myeloid leukemia: The challenges in diagnosis and monitoring minimal residual disease (MRD);Leukemia Research;2017-08
5. Chronic Myeloid Leukemia With Rare Variant b2a3 (e13a3) BCR-ABL1 Fusion;Annals of Laboratory Medicine;2016-05-01
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