ORMDL mislocalization by impaired autophagy in Niemann-Pick type C disease leads to increased de novo sphingolipid biosynthesis-Reference-Cited by-同舟云学术

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ORMDL mislocalization by impaired autophagy in Niemann-Pick type C disease leads to increased de novo sphingolipid biosynthesis

Published:2024-06 Issue:6 Volume:65 Page:100556
ISSN:0022-2275
Container-title:Journal of Lipid Research
language:en
Short-container-title:Journal of Lipid Research

Author:

Brown Ryan D.R.^ORCID,Mahawar Usha,Wattenberg Binks W.,Spiegel Sarah^ORCID

Publisher

Elsevier BV

Reference56 articles.

1. Niemann-Pick diseases;Vanier;Handb. Clin. Neurol.,2013

2. Structure of N-terminal domain of NPC1 reveals distinct subdomains for binding and transfer of cholesterol;Kwon;Cell,2009

3. NPC2 facilitates bidirectional transfer of cholesterol between NPC1 and lipid bilayers, a step in cholesterol egress from lysosomes;Infante;Proc. Natl. Acad. Sci. U. S. A.,2008

4. NPC intracellular cholesterol transporter 1 (NPC1)-mediated cholesterol export from lysosomes;Pfeffer;J. Biol. Chem.,2019

5. Free sphingoid bases in tissues from patients with type C Niemann-Pick disease and other lysosomal storage disorders;Rodriguez-Lafrasse;Biochim. Biophys. Acta,1994

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