Differentiation of renal Na+-K+-ATPase in control and streptozotocin-induced diabetic mice by G-protein acting toxins and phorbol esters-Reference-Cited by-同舟云学术

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Differentiation of renal Na+-K+-ATPase in control and streptozotocin-induced diabetic mice by G-protein acting toxins and phorbol esters

Published:1992-04 Issue:4 Volume:225 Page:275-279
ISSN:0922-4106
Container-title:European Journal of Pharmacology: Molecular Pharmacology
language:en
Short-container-title:European Journal of Pharmacology: Molecular Pharmacology

Author:

Chen Ching-Chow

Publisher

Elsevier BV

Subject

Pharmacology

Reference32 articles.

1. Pertussis toxin modulates dopamine inhibition of Na, K-ATPase activity in rat proximal convulated tubule segments;Bertorello,1988

2. Regulation of Na+-K+-ATPase activity in kidney proximal tubules: involvement of GTP binding proteins;Bertorello;Am. J. Physiol.,1989

3. Na+-K+-ATPase is an effector protein for protein kinase C in renal proximal tubule cells;Bertorello;Am. J. Physiol.,1989

4. Development of increased renal Na+-K+-ATPase activity and glomerular filtration rate in the diabetic mice;Chen,1990

5. Mode of inhibitory action of on Na+-K+-ATPase activity of the rat synaptic membrane;Chen;Biochem. Pharmacol.,1985

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. C-Peptide as a Therapeutic Tool in Diabetic Nephropathy;American Journal of Nephrology;2010

2. Proinsulin C-peptide reduces diabetes-induced glomerular hyperfiltration via efferent arteriole dilation and inhibition of tubular sodium reabsorption;American Journal of Physiology-Renal Physiology;2009-11

3. The C-peptide fragment EVARQ reduces glomerular hyperfiltration in streptozotocin-induced diabetic rats;Diabetes/Metabolism Research and Reviews;2007

4. Transport Systems in Kidney Basolateral Membrane: Pathophysiologic Implications;Membrane Physiopathology;1994

5. Stimulation of alpha 2-adrenergic receptors increases Na(+)-K(+)-ATPase activity in distal convoluted tubule cells;American Journal of Physiology-Renal Physiology;1993-10-01

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