Mitochondria dysfunction and impaired response to oxidative stress promotes proteostasis disruption in aged human cells-Reference-Cited by-同舟云学术

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Mitochondria dysfunction and impaired response to oxidative stress promotes proteostasis disruption in aged human cells

Published:2023-03 Issue: Volume:69 Page:1-9
ISSN:1567-7249
Container-title:Mitochondrion
language:en
Short-container-title:Mitochondrion

Author:

Trigo Diogo,Nadais André,Carvalho Ana,Morgado Bárbara,Santos Francisco,Nóbrega-Pereira Sandrina,da Cruz e Silva Odete A.B.

Funder

Fundação para a Ciência e a Tecnologia

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Molecular Medicine

Reference43 articles.

1. Protein aggregation in E. coli : short term and long term effects of nutrient density;Baig;PLoS ONE,2014

2. Flavonoids as therapeutic compounds targeting key proteins involved in Alzheimer's disease;Baptista;ACS Chem. Neurosci.,2014

3. The Role of Catechins in Cellular Responses to Oxidative Stress;Bernatoniene;Molecules,2018

4. P62/SQSTM1 at the interface of aging, autophagy, and disease;Bitto;Age (Dordr),2014

5. Brain mitochondrial dysfunction in aging;Boveris;IUBMB Life,2008

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2. Towards Healthy Longevity: Comprehensive Insights from Molecular Targets and Biomarkers to Biological Clocks;International Journal of Molecular Sciences;2024-06-20

3. Unveiling the Journey from the Gut to the Brain: Decoding Neurodegeneration–Gut Connection in Parkinson’s Disease;ACS Chemical Neuroscience;2024-06-19

4. Loss of WDR23 proteostasis impacts mitochondrial homeostasis in the mouse brain;Cellular Signalling;2024-04

5. Redox imbalance and metabolic defects in the context of Alzheimer disease;FEBS Letters;2024-03-12

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